Quercetin Inflammation Meta-Analysis Evidence Table

Structured evidence table for Quercetin Inflammation Meta-Analysis, generated from 2 reusable source documents in the Migaku knowledge base.

topicclaimevidence levelcitationsource
Quercetin Inflammation Meta-AnalysisAlthough existing therapies, including corticosteroids, antivirals, and immunosuppressants, can delay progression of disease, they are often associated with high costs, side effects, and decreased long-term efficacy [].3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisFlavonoids comprise a structurally diverse group of naturally occurring polyphenolic compounds abundantly found in grains, tea, fruits, vegetables, and numerous medicinal plants [,,].3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisThe purpose of this review is to consolidate current mechanistic evidence of flavonoid-mediated redox regulation in liver disease, with particular emphasis on intracellular signalling pathways and gut–liver interactions, and to assess their potential therapeutic relevance.3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisIt plays a central role in maintaining the metabolism of lipids, proteins, and carbohydrates, thereby supporting overall physiological balance.3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisBackground Liver inflammation and fibrosis are directly associated with non-alcoholic fatty liver disease (NAFLD).4Alqahtani SA (2026)Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis.
Quercetin Inflammation Meta-AnalysisNAFLD was also associated with the upregulation of hepatic IL-1β, iNOS, CD45, and apoptosis (p53).4Alqahtani SA (2026)Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis.
Quercetin Inflammation Meta-AnalysisBiomarkers of fibrosis (TIMP-1 and α-SMA) were also elevated, and fibrosis was confirmed in the model group by increased collagen deposition and elevated stages of fibrosis score (Stage 1 to 2 of Brunt's NASH classification).4Alqahtani SA (2026)Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis.
Quercetin Inflammation Meta-AnalysisDysregulation of the potent pro-inflammatory cytokine interleukin-1 beta (IL-1β), inducible nitric oxide synthase (iNOS), and tissue leukocyte infiltration (CD45 +ve) are connected with multiorgan injury and fibrosis.4Alqahtani SA (2026)Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis.

Source documents

  1. Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
  2. Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis.