Quercetin Inflammation Meta-Analysis Evidence Table

Structured evidence table for Quercetin Inflammation Meta-Analysis, generated from 4 reusable source documents in the Migaku knowledge base.

topicclaimevidence levelcitationsource
Quercetin Inflammation Meta-AnalysisThis review was based on a narrative literature search designed to identify experimental, clinical, and translational evidence linking natural products with gut barrier integrity, gut microbiota, low-grade inflammation, and metabolic endotoxemia.3Šantić Roko (2026)Natural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
Quercetin Inflammation Meta-AnalysisBecause this article is a narrative review rather than a systematic review, formal risk-of-bias assessment and quantitative evidence synthesis were not performed.3Šantić Roko (2026)Natural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
Quercetin Inflammation Meta-AnalysisOver the past two decades, accumulating evidence has repositioned the gastrointestinal tract from a passive absorptive organ to an immunologically active barrier whose functional state directly influences systemic inflammatory tone and metabolic homeostasis [].3Šantić Roko (2026)Natural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
Quercetin Inflammation Meta-AnalysisReference lists of relevant reviews and primary studies were additionally screened to identify articles not captured by database searches.3Šantić Roko (2026)Natural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
Quercetin Inflammation Meta-AnalysisAlthough existing therapies, including corticosteroids, antivirals, and immunosuppressants, can delay progression of disease, they are often associated with high costs, side effects, and decreased long-term efficacy [].3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisFlavonoids comprise a structurally diverse group of naturally occurring polyphenolic compounds abundantly found in grains, tea, fruits, vegetables, and numerous medicinal plants [,,].3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisThe purpose of this review is to consolidate current mechanistic evidence of flavonoid-mediated redox regulation in liver disease, with particular emphasis on intracellular signalling pathways and gut–liver interactions, and to assess their potential therapeutic relevance.3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisIt plays a central role in maintaining the metabolism of lipids, proteins, and carbohydrates, thereby supporting overall physiological balance.3Dogra Shivani (2026)Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
Quercetin Inflammation Meta-AnalysisThese contributions, encompassing systematic reviews, meta-analyses, and experimental studies, collectively reflect a field that is evolving from descriptive exploration toward evidence-based and translational science.4Boylan Fabio (2026)Editorial: Plant polyphenols: new chemical and pharmacological approaches
Quercetin Inflammation Meta-AnalysisTheir findings consolidate evidence supporting curcumin’s antioxidant and anti-inflammatory roles in mitigating reproductive damage.4Boylan Fabio (2026)Editorial: Plant polyphenols: new chemical and pharmacological approaches
Quercetin Inflammation Meta-AnalysisTheir review highlights the capacity of these compounds to influence pathways associated with skeletal integrity and hormone-responsive tissues.4Boylan Fabio (2026)Editorial: Plant polyphenols: new chemical and pharmacological approaches
Quercetin Inflammation Meta-Analysis“Plant Polyphenols: New Chemical and Pharmacological Approaches” Plant polyphenols remain at the forefront of pharmacological research, offering a compelling interface between traditional medicinal knowledge and modern scientific innovation (1).4Boylan Fabio (2026)Editorial: Plant polyphenols: new chemical and pharmacological approaches
topicQuercetin Inflammation Meta-Analysis
claimThis review was based on a narrative literature search designed to identify experimental, clinical, and translational evidence linking natural products with gut barrier integrity, gut microbiota, low-grade inflammation, and metabolic endotoxemia.
evidence level3
citationŠantić Roko (2026)
sourceNatural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
topicQuercetin Inflammation Meta-Analysis
claimBecause this article is a narrative review rather than a systematic review, formal risk-of-bias assessment and quantitative evidence synthesis were not performed.
evidence level3
citationŠantić Roko (2026)
sourceNatural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
topicQuercetin Inflammation Meta-Analysis
claimOver the past two decades, accumulating evidence has repositioned the gastrointestinal tract from a passive absorptive organ to an immunologically active barrier whose functional state directly influences systemic inflammatory tone and metabolic homeostasis [].
evidence level3
citationŠantić Roko (2026)
sourceNatural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
topicQuercetin Inflammation Meta-Analysis
claimReference lists of relevant reviews and primary studies were additionally screened to identify articles not captured by database searches.
evidence level3
citationŠantić Roko (2026)
sourceNatural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
topicQuercetin Inflammation Meta-Analysis
claimAlthough existing therapies, including corticosteroids, antivirals, and immunosuppressants, can delay progression of disease, they are often associated with high costs, side effects, and decreased long-term efficacy [].
evidence level3
citationDogra Shivani (2026)
sourceRedox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
topicQuercetin Inflammation Meta-Analysis
claimFlavonoids comprise a structurally diverse group of naturally occurring polyphenolic compounds abundantly found in grains, tea, fruits, vegetables, and numerous medicinal plants [,,].
evidence level3
citationDogra Shivani (2026)
sourceRedox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
topicQuercetin Inflammation Meta-Analysis
claimThe purpose of this review is to consolidate current mechanistic evidence of flavonoid-mediated redox regulation in liver disease, with particular emphasis on intracellular signalling pathways and gut–liver interactions, and to assess their potential therapeutic relevance.
evidence level3
citationDogra Shivani (2026)
sourceRedox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
topicQuercetin Inflammation Meta-Analysis
claimIt plays a central role in maintaining the metabolism of lipids, proteins, and carbohydrates, thereby supporting overall physiological balance.
evidence level3
citationDogra Shivani (2026)
sourceRedox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
topicQuercetin Inflammation Meta-Analysis
claimThese contributions, encompassing systematic reviews, meta-analyses, and experimental studies, collectively reflect a field that is evolving from descriptive exploration toward evidence-based and translational science.
evidence level4
citationBoylan Fabio (2026)
sourceEditorial: Plant polyphenols: new chemical and pharmacological approaches
topicQuercetin Inflammation Meta-Analysis
claimTheir findings consolidate evidence supporting curcumin’s antioxidant and anti-inflammatory roles in mitigating reproductive damage.
evidence level4
citationBoylan Fabio (2026)
sourceEditorial: Plant polyphenols: new chemical and pharmacological approaches
topicQuercetin Inflammation Meta-Analysis
claimTheir review highlights the capacity of these compounds to influence pathways associated with skeletal integrity and hormone-responsive tissues.
evidence level4
citationBoylan Fabio (2026)
sourceEditorial: Plant polyphenols: new chemical and pharmacological approaches
topicQuercetin Inflammation Meta-Analysis
claim“Plant Polyphenols: New Chemical and Pharmacological Approaches” Plant polyphenols remain at the forefront of pharmacological research, offering a compelling interface between traditional medicinal knowledge and modern scientific innovation (1).
evidence level4
citationBoylan Fabio (2026)
sourceEditorial: Plant polyphenols: new chemical and pharmacological approaches

Source documents

  1. Natural Bioactive Compounds Targeting Gut Barrier Integrity and Metabolic Endotoxemia in Cardiometabolic Disease: Mechanistic Insights and Translational Perspectives
  2. Redox Reprogramming of the Diseased Liver by Dietary Flavonoids: From Molecular Signalling to Gut–Liver Crosstalk
  3. Editorial: Plant polyphenols: new chemical and pharmacological approaches
  4. Quercetin Attenuates Non-Alcoholic Fatty Liver Disease in Association with the Inhibition of Hepatic IL-1β/iNOS and IL-1β/CD45 Axes of Inflammation and Fibrosis Accompanied by Reduced Endogenous Metabolites and Apoptosis