Structured evidence table for Carnitine Cognitive Performance Meta-Analysis, generated from 2 reusable source documents in the Migaku knowledge base.
| topic | claim | evidence level | citation | source |
|---|---|---|---|---|
| Carnitine Cognitive Performance Meta-Analysis | 3 4 [5] [6] [7] [8] 9 10 Previous studies have associated dementia risk with socioeconomic status (SES), a comprehensive concept that measured a person’s position from income, education and occupation [,]. | 3 | Zhao Xingguang (2026) | Income, diet, and cognitive function: observational analyses and candidate metabolomic pathways identified by Mendelian randomization |
| Carnitine Cognitive Performance Meta-Analysis | Evidence suggests that SES may influence cognitive function through multiple mediating pathways, including lifestyle behaviors, depressive symptoms, and vascular risk factors [,,,], highlighting potential targets for interventions to reduce the burden of dementia. | 3 | Zhao Xingguang (2026) | Income, diet, and cognitive function: observational analyses and candidate metabolomic pathways identified by Mendelian randomization |
| Carnitine Cognitive Performance Meta-Analysis | Diet quality generally improved with income [], while low-income families tend to purchase unhealthy food products rather than intake of enough types and frequency of healthy foods including vegetables and fruits, neglecting the nutritional quality of foods []. | 3 | Zhao Xingguang (2026) | Income, diet, and cognitive function: observational analyses and candidate metabolomic pathways identified by Mendelian randomization |
| Carnitine Cognitive Performance Meta-Analysis | 1 2 Dementia, including Alzheimer's disease (AD), is one of the primary causes of disability and death among the older people, and represents a serious public health issue []. | 3 | Zhao Xingguang (2026) | Income, diet, and cognitive function: observational analyses and candidate metabolomic pathways identified by Mendelian randomization |
| Carnitine Cognitive Performance Meta-Analysis | Brain morphometric analysis showed diffused reduced volumes and cortical thinning, involving fronto-temporal areas and, to a lesser extent, the parieto-occipital regions. | 3 | Veronelli L (2026) | Brain morphometry and cognition in late-onset glutaric aciduria type 1: scoping review and novel insights from a case report. |
| Carnitine Cognitive Performance Meta-Analysis | CONCLUSION: The present review and case report suggest that integrating neuroanatomical and neuropsychological investigations into clinical practice may allow a more refined characterization of GA1 patients, contributing to unveil the complex ethio-pathogenic mechanisms underlying the disease and monitor patients over time. | 3 | Veronelli L (2026) | Brain morphometry and cognition in late-onset glutaric aciduria type 1: scoping review and novel insights from a case report. |
| Carnitine Cognitive Performance Meta-Analysis | INTRODUCTION: Glutaric Aciduria type I (GA1) is a rare autosomal recessive organic aciduria, with typical early-onset presentation, characterized by severe movement disorders with damage to the basal ganglia following an acute encephalopathic crisis. | 3 | Veronelli L (2026) | Brain morphometry and cognition in late-onset glutaric aciduria type 1: scoping review and novel insights from a case report. |
| Carnitine Cognitive Performance Meta-Analysis | In LO patients, severe brain damage is frequently reported, including frontotemporal hypoplasia, brain atrophy, subependymal nodules and leukodystrophy. | 3 | Veronelli L (2026) | Brain morphometry and cognition in late-onset glutaric aciduria type 1: scoping review and novel insights from a case report. |
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