Structured evidence table for Resistant Starch Gut Microbiome Meta-Analysis, generated from 2 reusable source documents in the Migaku knowledge base.
| topic | claim | evidence level | citation | source |
|---|---|---|---|---|
| Resistant Starch Gut Microbiome Meta-Analysis | 1 2 3 The condition now referred to as metabolic dysfunction-associated fatty liver disease has traversed a long and contested nosological path. | 3 | Wang Rui (2026) | Metabolic dysfunction-associated fatty liver disease: redefining the conceptual framework from nomenclature to emerging therapeutic paradigms |
| Resistant Starch Gut Microbiome Meta-Analysis | Throughout this review, we use the term MASLD (metabolic dysfunction-associated steatotic liver disease) as the current preferred nomenclature, while retaining the historical terms NAFLD and MAFLD when describing the evolution of the field or citing studies that employed those frameworks. | 3 | Wang Rui (2026) | Metabolic dysfunction-associated fatty liver disease: redefining the conceptual framework from nomenclature to emerging therapeutic paradigms |
| Resistant Starch Gut Microbiome Meta-Analysis | When Ludwig, Viggiano, McGill, and Oh introduced “non-alcoholic steatohepatitis” (NASH) in 1980, they described hepatic macrovesicular steatosis with lobular inflammation in patients whose alcohol consumption fell well below the threshold typically associated with alcoholic liver injury (). | 3 | Wang Rui (2026) | Metabolic dysfunction-associated fatty liver disease: redefining the conceptual framework from nomenclature to emerging therapeutic paradigms |
| Resistant Starch Gut Microbiome Meta-Analysis | Over four decades, what began as a histological description has evolved into one of the most extensively studied metabolic conditions of the 21st century, driven by the parallel global rise in obesity, type 2 diabetes, and sedentary lifestyles. | 3 | Wang Rui (2026) | Metabolic dysfunction-associated fatty liver disease: redefining the conceptual framework from nomenclature to emerging therapeutic paradigms |
| Resistant Starch Gut Microbiome Meta-Analysis | Reduced renal sodium excretion promotes salt and volume expansion, while sympathetic nervous system overactivity and activation of the renin–angiotensin–aldosterone system further increase vascular resistance, sodium retention, and blood pressure. | 3 | Agustin Maya (2026) | Fruits, Vegetables, and Legumes: Harnessing the Gut-Kidney Axis in Hypertensive Chronic Kidney Disease |
| Resistant Starch Gut Microbiome Meta-Analysis | Reviews of human and translational data describe increased urea diffusion into the gut lumen, disruption of intestinal epithelial integrity, greater permeability, altered motility, and a shift toward a proinflammatory intestinal milieu. | 3 | Agustin Maya (2026) | Fruits, Vegetables, and Legumes: Harnessing the Gut-Kidney Axis in Hypertensive Chronic Kidney Disease |
| Resistant Starch Gut Microbiome Meta-Analysis | Mechanistic evidence is separated, as far as possible, from evidence strong enough to support clinical recommendations. | 3 | Agustin Maya (2026) | Fruits, Vegetables, and Legumes: Harnessing the Gut-Kidney Axis in Hypertensive Chronic Kidney Disease |
| Resistant Starch Gut Microbiome Meta-Analysis | 1 2 3 3 Hypertension is central to the clinical burden of chronic kidney disease (CKD) [,]. | 3 | Agustin Maya (2026) | Fruits, Vegetables, and Legumes: Harnessing the Gut-Kidney Axis in Hypertensive Chronic Kidney Disease |
Source documents