Okra Blood Glucose Randomized Trial Evidence Table

Structured evidence table for Okra Blood Glucose Randomized Trial, generated from 1 reusable source document in the Migaku knowledge base.

topicclaimevidence levelcitationsource
Okra Blood Glucose Randomized TrialDiabetes induction led to decreased body weight, increased blood glucose levels.4Alblihd MA (2023)Okra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
Okra Blood Glucose Randomized TrialCapsular thickness was significantly increased, white pulp was widely dispersed, and mature lymphocytes in the periphery were also drastically decreased, with thick follicular arteries, necrosis, and depletion of lymphocytes in the germinal center.4Alblihd MA (2023)Okra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
Okra Blood Glucose Randomized TrialDiabetes mellitus is a complex metabolic syndrome that involves dysfunction of spleen and other lymphoid organs.4Alblihd MA (2023)Okra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
Okra Blood Glucose Randomized TrialMedicinal plants, including okra ( Abelmoschus esculentus (L.) Moench), were used widely for diabetes treatment.4Alblihd MA (2023)Okra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
topicOkra Blood Glucose Randomized Trial
claimDiabetes induction led to decreased body weight, increased blood glucose levels.
evidence level4
citationAlblihd MA (2023)
sourceOkra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
topicOkra Blood Glucose Randomized Trial
claimCapsular thickness was significantly increased, white pulp was widely dispersed, and mature lymphocytes in the periphery were also drastically decreased, with thick follicular arteries, necrosis, and depletion of lymphocytes in the germinal center.
evidence level4
citationAlblihd MA (2023)
sourceOkra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
topicOkra Blood Glucose Randomized Trial
claimDiabetes mellitus is a complex metabolic syndrome that involves dysfunction of spleen and other lymphoid organs.
evidence level4
citationAlblihd MA (2023)
sourceOkra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.
topicOkra Blood Glucose Randomized Trial
claimMedicinal plants, including okra ( Abelmoschus esculentus (L.) Moench), were used widely for diabetes treatment.
evidence level4
citationAlblihd MA (2023)
sourceOkra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.

Source documents

  1. Okra [<i>Abelmoschus esculentus</i> (L.) Moench] improved blood glucose and restored histopathological alterations in splenic tissues in a rat model with streptozotocin-induced type 1 diabetes through CD8<sup>+</sup> T cells and NF-kβ expression.